Acid-Base Regulation

by Shahid, Asma

Physiology

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Slzcqxujs4gzmssh1qyu 180827 s0 shahid asma acid base regulation intro
02:06
Acid-Base Regulation
Zlslzxjat4yia5yd13nr 180827 s1 shahid asma acids and bases
14:03
Acids and Bases
Jpbgmykdtri6rfowcjip 180827 s2 shahid asma buffer systems
20:20
Buffer Systems
Gc2q2lsiqqvr50zhvvmi 180827 s3 shahid asma renal control of acid base balance
18:52
Renal Control of Acid-Base Balance
Q5dipfotqgvix1qfnwtg 180827 s4 shahid asma quantifying renal acid base excretion
07:12
Quantifying Renal Acid-Base Excretion
Eylpqbxar7usegwwmlir 180827 s5 shahid asma clinical causes of acid base disorders
06:59
Clinical Causes of Acid Base Disorders

Lecture´s Description

Acids and Bases
In this lecture ´´Acid-Base Regulation´´ are explained. Section one is about ´´Acids and Bases´´. Educator's prime focus is explanation of Regulation of H+. After that discusses acids and bases. Then elucidates strong and weak acids and bases. Following this, normal H+ concentration and pH of body fluids is presented. Furthermore, buffers, lungs and kidneys are pursued. Lastly, buffering of H+ in the body fluid is highlighted.


Buffer Systems
Section two is about ‘’Buffer Systems’’. Initially, bicarbonate buffer system is elaborated. Then quantitative dynamics of HCO3- buffer system are discussed. This followed by phosphate buffer system. After that proteins as intracellular buffers is explained. Likewise, respiratory regulation of acid-base balance is pursued. Moving onward, information is conveyed about expiration of CO2 balances formation of CO2. Then alveolar ventilation is elucidated. Later on, feedback control of H+ concentration is discussed. Educator also talks about efficiency of respiratory control. At last, respiratory acidosis is exhibited.


Renal Control of Acid-Base Balance
Section three is about ‘’Renal Control of Acid-Base Balance’’. In the beginning, educator discussed renal control of acid-base balance. After that renal secretion of H+ and reabsorption of HCO3− is pursued. Then information is conveyed about H+ secreted in the early tubular segments. Alongside, educator tells that filtered HCO3- is reabsorbed. Following this, secretion of H+ in late distal and collecting tubules is focused. Furthermore, combination of excess H+ with PO3- and NH3 buffers is reported. Next subject of discussion in this section is that phosphate buffer system generates new HCO3−. Subsequently, ammonia buffer system is elucidated. Lastly, discussion continues on chronic acidosis increases NH4+ excretion.


Quantifying Renal Acid-Base Excretion
Section four is about ‘’Quantifying Renal Acid-Base Excretion’’. Firstly, quantifying renal acid-base excretion is discussed. After that regulation of renal tubular H+ secretion is explained. Then renal correction of acidosis is pursued. Moreover, educator tells that acidosis decreases HCO3−/H+ ratio. Subsequently, renal correction of alkalosis is elaborated. Lastly, HCO3−/H+ ratio in renal tubular fluid is highlighted.


Clinical Causes of Acid Base Disorders
Section five is about ‘’Clinical Causes of Acid Base Disorders’’. Educator begins by explaining clinical causes of metabolic acidosis. Then moves to the discussion of clinical causes of metabolic alkalosis.  Following this, comprehensive information about treatment of acidosis or alkalosis is given.

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