Your browser is too old
We can't provide a great video experience on old browserUpdate now
Subdural empyema is the cornerstone of this sqadia.com medical video lecture. Starting off with epidemiology and pathophysiology of subdural empyema, clinical presentation and diagnosis also comes under debate. Subsequently, treatment and prognosis are pursued along with intracranial epidural abscess. Conclusively, suppurative thrombophlebitis is considered.
Epidemiology and Pathophysiology
Subdural empyema (SDE) is a collection of pus between dura and arachnoid membranes. It is a rare disorder and epidemiologically accounts for 15-25%. Etiology of subdural empyema include aerobic and anaerobic streptococci, staphylococci, Enterobacteriaceae. Pathophysiology indicate it can be due to retrograde spread of infection, contiguous spread of infection, and direct introduction of bacteria.
Clinical Presentation and Diagnosis
Fever, progressively worsening headache, seizures, nuchal rigidity are the clinical presentations. Seizures begin as partial motor seizures. MRI is indicated for diagnosis. Moreover, administration of gadolinium greatly improves diagnosis. CT Scan shows crescent-shaped hypodense lesion. Likewise, information about differential diagnosis is also delivered.
Treatment and Prognosis
Emergent neurosurgical evacuation of the empyema and empirical antimicrobial therapy are the treatment approaches. Moreover, combination therapy is also used. For treatment of hospital-acquired SDE, coverage with carbapenem and vancomycin is advised. Lastly, prognosis is discussed. Long-term neurologic sequelae occur in 50% cases.
Intracranial Epidural Abscess
A suppurative infection occurring in potential space between inner skull table and dura is referred as intracranial epidural abscess. Etiology and pathophysiology is deliberated in succession. Diagnosis is considered when fever, headache follow recent head trauma. Alongside, light is shed o treatment and prognosis.
A septic venous thrombosis of cortical veins and sinuses is known as suppurative thrombophlebitis. After that, Dr. Saima Mushtaq talks about anatomy and pathophysiology. Infection spreads from mastoid air cells to transverse sinus. Clinical manifestations include weakness of lower extremities. Clinical manifestations of septic cavernous sinus thrombosis and transverse sinus thrombosis are also enlightened.